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The B-cell antigen receptor (BCR) is expressed in B-cells prior to the onset of antigen receptor rearrangement. In contrast to T-cells, B-cell precursors do not undergo terminal differentiation into plasma cells in response to antigen. In this study, we demonstrate that B-cells respond differently than T-cells by downregulating bcl2 expression when exposed to antigen. Moreover, this effect is independent of B-cell maturation. Surprisingly, the bcl2 gene product does not inhibit apoptosis in B-cells or their precursors. Instead, B-cells undergoing apoptosis are rescued by the bcl2 gene product and their progeny is rescued as well. The ability of B-cells to survive long-term in the absence of the bcl2 gene 0b46394aab